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Asthma is a chronic inflammatory disease of airways that is characterized by increased responsiveness of the trachea bronchial tree to a multiplicity of stimuli. It is manifested physiologically by a widespread narrowing of the air passage and clinically by paroxysms of dyspnea, cough and wheezing.
Atopy is the single largest risk factor for the development of asthma. Allergic asthma is often associated with a personal and or family history of allergic disease such as rhinitis.
The prevalence of asthma is estimated to be 4 to 5 % of the world population.
The physiological and clinical features of asthma derive from an interaction among infiltrating inflammatory molecules in the airway surface epithelium, inflammatory Mediators and Cytokines. Thus Asthma is a result of a chronic inflammatory process.
Indus is focusing on an interesting target of inflammation viz. Phosphodiesterase 4 enzyme. The Indus candidate has very low or nil emetic potential and other usual side effects of this class of Molecules.
Atopy is the single largest risk factor for the development of asthma. Allergic asthma is often associated with a personal and or family history of allergic disease such as rhinitis.
The prevalence of asthma is estimated to be 4 to 5 % of the world population.
The physiological and clinical features of asthma derive from an interaction among infiltrating inflammatory molecules in the airway surface epithelium, inflammatory Mediators and Cytokines. Thus Asthma is a result of a chronic inflammatory process.
Indus is focusing on an interesting target of inflammation viz. Phosphodiesterase 4 enzyme. The Indus candidate has very low or nil emetic potential and other usual side effects of this class of Molecules.
